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Collated from TripSit, Pharmacology, DrugCentral. Where sources differ (e.g. dosing), Compare shows them side by side.

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Is a barbiturate that was once used for inducing anesthesia for surgery. It has a very rapid onset, and a short duration of effects.TS

Oral

Route dataTripSit

ThresholdLightCommonStrongHeavy
10–15 mg15–20 mg20–30 mg—+
030 mg
LightCommonStrongHeavy
Onset1–2 minutes
Total30–45 minutes
After-effects1–4 hours
OnsetCome-upPeakOffset

🧬 Receptor activityDC

TargetActionAffinitySource
GABA-A receptor alpha-1/beta-3/gamma-2 (GABRA1)DRUGCENTRAL
GABA-A receptor alpha-2/beta-3/gamma-2 (GABRG2)DRUGCENTRAL
GABA-A receptor alpha-3/beta-3/gamma-2 (GABRG2)DRUGCENTRAL
GABA-A receptor alpha-5/beta-3/gamma-2 (GABRG2)DRUGCENTRAL
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Mechanism of actionPH

Hexobarbital binds at a distinct binding site associated with a Cl- ionopore at the GABA-A receptor, increasing the duration of time for which the Cl- ionopore is open. The post-synaptic inhibitory effect of GABA in the thalamus is, therefore, prolonged.

PharmacodynamicsPH

Hexobarbital is a barbiturate derivative having hypnotic and sedative effects. It was subsequently used in the 1940s and 1950s as an anesthetic for surgery. Furthermore, the agent also demonstrates a fairly quick onset of action that also possesses a short duration of action. However it can be difficult to control the depth of anesthesia with hexobarbital which makes it quite dangerous, and it has now been replaced by safer drugs in human medicine, usually thiopental would be the barbiturate of choice for this application these days.

Pharmacokinetics

MetabolismPH

Hepatic.

Protein bindingPH

25%

Plan when to take Hexobarbital — see where onset, peak and comedown land on the clock

Fact-sheets from PsychonautWiki. Harm-reduction reference only — not medical advice.