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Collated from TripSit, Pharmacology, DailyMed, PiHKAL. Where sources differ (e.g. dosing), Compare shows them side by side.

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Also known as 2,5-DIMETHOXY-4-(n)-PROPYLAMPHETAMINEPI

A rare psychedelic amphetamine of the same class as DOM. This is a powerful and potent psychedelic with all the effects you would expect from an amphetamine. Described by Shulgin as a 'heavy duty psychedelic'. Analogue of 2C-P.TS

Oral

Route dataTripSit

ThresholdLightCommonStrongHeavy
1.25 mg2.5–3.5 mg3.5 mg+
04.375 mg
LightCommonStrongHeavy
Onset90–240 minutes
Total18–30 hours
After-effects1–24 hours
OnsetCome-upPeakOffset

🧬 Receptor activityPH

TargetActionAffinitySource
5-hydroxytryptamine receptor 2CKi 14 nMCHEMBL
Serotonin 2 (5-HT2) receptorKi 69 nMCHEMBL
Serotonin 1 (5-HT1) receptorKi 3170 nMCHEMBL
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PharmacodynamicsDM

Doxapram hydrochloride produces respiratory stimulation mediated through the peripheral carotid chemoreceptors. As the dosage level is increased, the central respiratory centers in the medulla are stimulated with progressive stimulation of other parts of the brain and spinal cord.
The onset of respiratory stimulation following the recommended single intravenous injection of doxapram hydrochloride usually occurs in 20 to 40 seconds with peak effect at 1 to 2 minutes. The duration of effect may vary from 5 to 12 minutes.
The respiratory stimulant action is manifested by an increase in tidal volume associated with a slight increase in respiratory rate.
A pressor response may result following doxapram administration. Provided there is no impairment of cardiac function, the pressor effect is more marked in hypovolemic than in normovolemic states. The pressor response is due to the improved cardiac output rather than peripheral vasoconstriction. Following doxapram administration, an increased release of catecholamines has been noted.
Although opiate-induced respiratory depression is antagonized by doxapram, the analgesic effect is not affected.

Pharmacokinetics

AbsorptionDM

Doxapram is metabolized via ring hydroxylation to ketodoxapram, an active metabolite readily detected in the plasma.

Plan when to take DOPR — see where onset, peak and comedown land on the clock

Fact-sheets from PsychonautWiki. Harm-reduction reference only — not medical advice.